Heart attack Case Study Example | Topics and Well Written Essays - 500 words

Heart attack - Case Study Example Lack of this results in the death of its muscle cells which brings about heart attack. This disorder is also referred to as a myocardial infarction as it results in a permanent death of the heart muscle. ‘Myo’ means muscle, ‘cardial’ is the heart while ‘infaction’ refers to the death of a tissue due to lack of blood supply. A possible remedy for this disorder is angioplasty. This refers to a surgical unblocking or repair of the coronary artery. This surgical procedure is often done to open up blocked or narrow coronary arteries and reinstate the flow of blood to the heart muscle. A coronary stent is a small, expandable tube that is used in the treatment of narrowed or blocked coronary artery. This tube is placed in the coronary artery to keep it open so as to ensure a constant flow of blood to the heart muscle. Coronary stents are placed in the affected coronary artery during a procedure referred to as angioplasty or percutaneous coronary interv ention (PCI). Coronary stents treat heart attack disorder as they ensure that the coronary artery remains open. This ensures that there is a constant and continuous flow of blood to the heart muscles through the coronary artery. Insertion of a coronary stent is considered to be minimally invasive as it does not involve any major incisions. It is done within a short period of time and the patients undergo less discomfort as compared to other heart procedures. Additionally, it only takes a short period of time for the patient to achieve full recovery.

Case study reflection 2 pages per case 8 total Essay

Case study reflection 2 pages per case 8 total - Essay Example tudy of metformin-associated lactic acidosis-MALA, in which the interactions of drug that is, orlistat in the long run and cimetidine in the short run, may have triggered the woman’s condition (British Medical Association, 1988, pp56-87). The case involves a 59-year-old woman having had diabetes type 2 for 14 years, presented a history of 3 months of unclear abdominal pain and 4-5 daily loose movements of bowel. Her condition had worsened for over 4 days before her admission to the hospital. On the admission day, she exhibited signs of weakness, dizziness, and blurred vision. In addition, her husband had observed inaudible speech and an abridged level of consciousness (Ehrman, 2009, pp234-345). When the diagnosis of metformin-associated lactic acidosis with cardiovascular collapse and acute prerenal renal failure was made, it was discovered that she needed She required a vigorous rehydration, infusion of sodium bicarbonate, support of inotropic, and the therapy of renal replacement. Empirically, all cultures of blood, urine, and feces were sterile. Three years later she was dialysis independent and the stability of became real having a creatinine of 250 ÃŽ ¼mol/l. The study aims at answering the question of what initiates MALA in patients who previously had normal renal functions. Though the approach of handling this condition is unknown, the available options have been supportive and usually focus stopping the drug, correcting the acidosis and treating the coexisting conditions which in most instances are renal impairment. The therapy of renal impairment eliminates lactate and metformin from the blood. Metformin is absorbed comparatively rapidly in the intestines, and is not metabolized. And 90% of the drug is removed through glomerular filtration and secretions in the tubules. It has a half-life of 1.5-5 hours (Dong, 2006, pp34-45). When compared with phenformin, it yields a negligible increase in producing lactate, which seems to be past the extra hepatic